Table of Contents
- 1 What can interfere with cellular respiration?
- 2 What stage of cellular respiration is affected by the poison?
- 3 How does arsenic interfere with cellular respiration?
- 4 What happens if cellular respiration is inhibited?
- 5 How would dinitrophenol poisoning affect electron transport and ATP synthesis?
- 6 How do respiratory poisons such as carbon monoxide and cyanide exert their effect?
- 7 How does carbon dioxide affect cellular respiration?
- 8 What happens to cellular respiration when there is poison in it?
- 9 What happens when a poison blocks the electron transport chain?
- 10 What poisons can be used to study oxygen consumption by mitochondria?
What can interfere with cellular respiration?
We’ll first review what cellular respiration is, and then explore how three factors affect it: temperature, glucose availability, and oxygen concentration.
What stage of cellular respiration is affected by the poison?
Cyanide poisons the mitochondrial electron transport chain within cells and renders the body unable to derive energy (adenosine triphosphate—ATP) from oxygen. Specifically, it binds to the a3 portion (complex IV) of cytochrome oxidase and prevents cells from using oxygen, causing rapid death.
What are the 3 major reactions in cellular respiration?
The reactions of cellular respiration can be grouped into three stages: glycolysis (stage 1), the Krebs cycle, also called the citric acid cycle (stage 2), and electron transport (stage 3).
How does arsenic interfere with cellular respiration?
Arsenic impairs cellular respiration by inhibiting various mitochondrial enzymes, and the uncoupling of oxidative phosphorylation. Toxic by-products are released when arsenic interacts with sulfhydryl groups of proteins and enzymes, and substitutes phosphorus in a variety of biochemical reactions [72].
What happens if cellular respiration is inhibited?
Without the process of cellular respiration, there is no gaseous exchange and the cells, tissue and other organs die due to the lack of oxygen and by the accumulation of carbon dioxide within the cells and tissues.
Which kind of metabolic poison would most directly interfere with glycolysis?
c
The kind of poison that would most likely directly interfere with glycolysis would be c. A poison that closely mimics the structure of glucose but is not metabolized. If the poison cannot be metabolized, then no NADH, pyruvate or ATP will be produced in glycolysis.
How would dinitrophenol poisoning affect electron transport and ATP synthesis?
This gradient of H+ can produce ATP by flowing through ATP synthetase in the mitochondrial inner membrane. Dinitrophenol disrupts the H+ gradient reducing ATP synthesis. Under these conditions, much of our food that we eat could not be used for ATP synthesis are we lose weight.
How do respiratory poisons such as carbon monoxide and cyanide exert their effect?
How does cyanide and Carbon monoxide effect cellular respiration? By binding the last protein of the electron transport chain to prevent it from transferring.
Does glyphosate inhibit cellular respiration?
Glyphosate also affected cell respiration, mainly inhibiting it, as shown in Fig. 1.
How does carbon dioxide affect cellular respiration?
During the process of cellular respiration, carbon dioxide is given off as a waste product. This carbon dioxide can be used by photosynthesizing cells to form new carbohydrates. Also in the process of cellular respiration, oxygen gas is required to serve as an acceptor of electrons.
What happens to cellular respiration when there is poison in it?
Poison in cellular respiration. When a poison such as cyanide blocks the electron transport chain during cellular respiration, glycolysis and the krebs cycle soon grind to halt as well.
What happens when cyanide is placed in cellular respiration?
Poison in cellular respiration. When a poison such as cyanide blocks the electron transport chain during cellular respiration, glycolysis and the krebs cycle soon grind to halt as well. Which of the following is the best explanation for this?
What happens when a poison blocks the electron transport chain?
When a poison such as cyanide blocks the electron transport chain during cellular respiration, glycolysis and the krebs cycle soon grind to halt as well.
What poisons can be used to study oxygen consumption by mitochondria?
Here is a list of poisons that can be used in the study of oxygen consumption by mitochondria, including sources and considerations for their use. Electron transport inhibitors Rotenone Antimycin Cyanide Malonate (succinate dehydrogenase inhibitor) Uncoupling agents 2,4-Dinitrophenol (DNP)